Ahmad Zrik, M.D.

Biography

Ahmad Zrik, M.D., is an Assistant Professor in the Department of Pediatrics at UT Southwestern Medical Center. He specializes in child neurology and pediatric epilepsy, with a focus on drug-resistant epilepsy, genetic epilepsies, advanced presurgical evaluation and treatment, and invasive EEG monitoring.

Dr. Zrik earned his medical degree at the University of Jordan. He completed residencies in pediatrics and child neurology at the University at Buffalo’s John R. Oishei Children’s Hospital, followed by an advanced fellowship in pediatric epilepsy at Texas Children’s Hospital in Houston.

Prior to his residencies, Dr. Zrik also completed specialized training in epilepsy at the University of Illinois Hospital and Clinics; clinical neurophysiology and pediatric epilepsy at Children’s Hospital of Michigan; and clinical research in neurophysiology at University Hospitals Cleveland Medical Center.

Certified by the American Board of Psychiatry and Neurology with a special qualification in child neurology and by the American Board of Clinical Neurophysiology in epilepsy monitoring and critical care EEG, he joined the UT Southwestern Division of Child Neurology faculty in 2026.

Dr. Zrik’s clinical expertise encompasses the comprehensive evaluation and management of pediatric epilepsy, including genetic epilepsies, drug-resistant epilepsy, epilepsy surgery evaluation, neuromodulation, and invasive EEG monitoring. He has a particular interest in advanced presurgical evaluation and surgical treatment of drug-resistant epilepsy.

He treats a wide range of conditions, including:

Complex and treatment-resistant syndromes

• Drug-resistant epilepsy
• Infantile epileptic spasms syndrome (West syndrome)
• Lennox-Gastaut syndrome
• Dravet syndrome and SCN1A-related epilepsies
• Developmental and epileptic encephalopathy with spike-wave activation in sleep (DEE-SWAS), including Landau-Kleffner syndrome
• Rasmussen encephalitis

Genetic and metabolic epileptic encephalopathies

• Early-onset developmental and epileptic encephalopathies, including Ohtahara syndrome and early myoclonic encephalopathy
• Monogenic epilepsies, including KCNQ2-, STXBP1-, SYNGAP1-, and CDKL5-related disorders
• Tuberous sclerosis complex-associated epilepsy
• Mitochondrial and metabolic epilepsies
• Glucose transporter type 1 deficiency syndrome (GLUT1 deficiency)

Structural, vascular, and autoimmune epilepsies

• Focal cortical dysplasia
• Hypothalamic hamartoma-associated epilepsy, including gelastic seizures
• Autoimmune-associated epilepsy and autoimmune encephalitis, including limbic encephalitis
• Epilepsy associated with stroke and perinatal brain injury

His academic interests focus on genetic epilepsy and precision medicine research, as well as advanced epilepsy surgery and invasive EEG research.